PCOS: Pathophysiology, Symptoms, and Diagnosis in Females
Abstract
Polycystic Ovary Syndrome (PCOS) is the most common endocrine disorder affecting women of reproductive age, with a prevalence ranging from 4 to 12 percent in different population studies. It is a heterogeneous disorder etiology of which is not known completely. Polycystic ovarian syndrome (PCOS) arises from ovarian dysfunction and various biochemical irregularities, serving as the leading cause of anovulatory infertility. Women affected by this condition typically exhibit characteristics such as infrequent or absent ovulation, signs of elevated androgens, the presence of multiple ovarian cysts, and an increased likelihood of experiencing dyslipidemia and insulin resistance. These metabolic issues contribute to long-term health concerns, accentuating the significance of addressing and managing PCOS for the well-being of affected individuals.
References
David Guzick, Polycystic ovary syndrome: Symptomatology, pathophysiology and epidemiology, Department of Obstetrics and Gynaecology, University of Rochester, 1998, pp 90.
Saad A K Amer, Polycystic ovarian syndrome, diagnosis and management of Infertility, Obstetrics, Gynecology and Reproductive medicine article, Elsevier, 2009, pp 264.
Marzieh Salehi, Rafael Bravo Vera, Arsalan Sheikh, Alina Gouller and Leonid Poretsky, pathogenesis of PCOS: what is the role of obesity article, Elsevier, 2004, pp 359.
Stein IF, Leventhal MI, amenorrhoea associated with bilateral polycystic ovaries,
American Journal of Obstetrics & Gynaecology, 1935, pp 181 .
Clinical gynecologic Endocrinology and Infertility, 7th edition, by Leon Speroff et al;
Jaypee Brothers, 2005. Part 1, chapter 6, page no. 187-221 / Krupa R Donga- Nasya and
Matra Basti by Narayana Taila on Anovulatory factor, 2010, IPGT&RA, GAU, Jamnagar.
Williams gynaecology(pdf), Third edition, Editors-Barbara L. Hoffman, MD; John O. Schorge, MD; Karen D. Bradshaw, MD; Lisa M. Halvorson, MD; Joseph I. Schaffer, MD; Marlene M. Corton, MD; Chapter 17.
Greenspans basic and clinical endocrinology, tenth edition, Edited by David G. Gardner, MD, MS ; Dolores Shoback, MD; Chapter 13, page 467.
Greenspans basic and clinical endocrinology, tenth edition, Edited by David G. Gardner, MD, MS ; Dolores Shoback, MD; Chapter 13, page 472. 9.Greenspans basic and clinical endocrinology, tenth edition, Edited by David G. Gardner,
MD, MS ; Dolores Shoback, MD; Chapter 13, page 472
Williams gynaecology(pdf), Third edition, Editors-Barbara L. Hoffman, MD; John O. Schorge, MD; Karen D. Bradshaw, MD; Lisa M. Halvorson, MD; Joseph I. Schaffer, MD; Marlene M. Corton, MD; Chapter 20, page 450.
Downloads
Published
Issue
Section
License
Declaration and Copyright Transfer Form
(to be completed by authors)
I/ We, the undersigned author(s) of the submitted manuscript, hereby declare, that the above manuscript which is submitted for publication in the STM Journals(s), is not published already in part or whole (except in the form of abstract) in any journal or magazine for private or public circulation, and, is not under consideration of publication elsewhere.
- I/We will not withdraw the manuscript after 1 week of submission as I have read the Author Guidelines and will adhere to the guidelines.
- I/We Author(s ) have niether given nor will give this manuscript elsewhere for publishing after submitting in STM Journal(s).
- I/ We have read the original version of the manuscript and am/ are responsible for the thought contents embodied in it. The work dealt in the manuscript is my/ our own, and my/ our individual contribution to this work is significant enough to qualify for authorship.
- I/We also agree to the authorship of the article in the following order:
Author’s name
1. ________________
2. ________________
3. ________________
4. ________________
| We Author(s) tick this box and would request you to consider it as our signature as we agree to the terms of this Copyright Notice, which will apply to this submission if and when it is published by this journal. |
